Volume 2, Issue 4 (2023)                   GMJM 2023, 2(4): 143-148 | Back to browse issues page
Article Type:
Analytical Review |
Subject:

Print XML PDF HTML


History

How to cite this article
Saeidi M, Mohammadkhani Orouji F. Symbolic Presence of Color. GMJM 2023; 2 (4) :143-148
URL: http://gmedicine.de/article-2-207-en.html
Download citation:
BibTeX | RIS | EndNote | Medlars | ProCite | Reference Manager | RefWorks
Send citation to:

Rights and permissions
1- Department of Psychology, Monireh Hospital of Medical Sciences, Tehran, Iran
2- Departmentof Psychology, Abarkouh Branch, Islamic Azad University, Abarkouh, Iran
* Corresponding Author Address: Departmentof Psychology, Abarkouh Branch, Islamic Azad University, Abarkouh, Iran. (f.mohammadkhani.or1983@gmail.com)
Abstract   (535 Views)
Introduction: “Symbol" is a medium that refers the audience to a meaning beyond itself. Now there may be an intrinsic connection between the mediator and the meaning that is associated, and it is likely that there is no pre-existing connection between the two. The first possibility belongs to natural symbols derived from the womb of life, phenomena and relationships, and the second possibility belongs to completely conventional symbols. Blue is a natural symbol of our experience and interaction with the sky. In front of the narrow eyes of man, there is always a sky with infinite vastness and every day of life he witnesses the unsuccessful attempt of the eyes to swallow this boundless vastness in such a way that the more you look the more you will find the limitedness of the eyes and the infinity of the sky. This is why blue is considered a symbol of infinity, and here there is an inherent relationship between the symbol and the relevant meaning. The red color, when present in the traffic light, evokes the meaning of stopping.
Conclusion: Such a symbol is merely a contract and a joint appointment made from a certain date between a group of people related to the subject, without any historical background.
 
Keywords:
|   |   Full-Text (HTML)  (136 Views)  

References
1. Kannan K, Jain SK. Oxidative stress and apoptosis. Pathophysiology. 2000;7(3):153-63. [Link] [DOI:10.1016/S0928-4680(00)00053-5]
2. Kapur I, Macdonaald RL. Rapid Seizure-Induced Reduction of Benzodiazepine and Zn2+ Sensitivity of Hippocampal Dentate Granule Cell GABAA Receptors. J Neurosci. 1997;17(19):153-4. [Link] [DOI:10.1523/JNEUROSCI.17-19-07532.1997]
3. Kaputlu I, Uzbay T. l-NAME inhibits pentylenetetrazole and strychnine-induced seizures in mice. Brain Res. 1997;753(1):98-101. [Link] [DOI:10.1016/S0006-8993(96)01496-5]
4. Kaviarasan K, Arjunan MM, Pugalendi KV. Lipid profile, oxidant-antioxidant status and glycoprotein components in hyperlipidemic patients with/without diabetes. Clin Chim Acta. 2005;362(1-2):49-56. [Link] [DOI:10.1016/j.cccn.2005.05.010]
5. Khine H, Weiss D, Graber N, Hoffman RS, Esteban-Cruciani N, Avner JR. A Cluster of children with seizures caused by camphor poisoning. Pediatrics. 2009;123(5):1269-72. [Link] [DOI:10.1542/peds.2008-2097]
6. Kilian M, Freg HH. Central monoamines and convulsive thresholds in mice and rats. Neurpharmacology. 1973;12(7):681-92. [Link] [DOI:10.1016/0028-3908(73)90121-4]
7. King H, Aubert RE, Herman WH. Global burden of Diabetes, 1995-2025: Prevalence, numerical estimates, and projections. Diabetes Care. 1998;21(9):1414-31. [Link] [DOI:10.2337/diacare.21.9.1414]
8. Janusz W, Kleinork Z. The role of the central serotonergic system in pilocarpine-induced seizures: receptor mechanisms. Neurosci Res. 1989;7(2):144-53. [Link] [DOI:10.1016/0168-0102(89)90054-0]
9. Kley S, Caffall Z, Tittle E, Ferguson DC, Hoenig M. Development of a feline proinsulin immunoradiometric assay and a feline proinsulin enzyme-linked immunosorbent assay (ELISA): A novel application to examine beta cell function in cats. Domest Anim Endocrinol. 2008;34(3):311-8. [Link] [DOI:10.1016/j.domaniend.2007.09.001]
10. Knekt P, Beunanen A, Jarvinen R, Seppane R, Helio VM, Aroma A. Antioxidant vitamin intake and coronary mortality in a longitudinal population study. Am J Epidemiol. 1994;139(13):1180-9. [Link] [DOI:10.1093/oxfordjournals.aje.a116964]
11. Kumarasamy Y, Nahar L, Byres M, Delazar A, Sarker SD. The assessment of biological activities associated with the major constituents of the methanol extract of 'wild carrot' (Daucus carotaL.) seeds. J Herb Pharmacother. 2005;5(1):61-72. [Link] [DOI:10.1300/J157v05n01_07]
12. Kuzuya T, Nakagawa S, Satoh J, Kanazawa S, Iwamota Y, Kobayashi M, et al. Report of the Committee on the classification and diagnostic criteria of diabetes mellitus. Diabetes Res Clin Pract. 2002;55(1):65-85. [Link] [DOI:10.1016/S0168-8227(01)00365-5]
13. Lee J, Sparrow D, Vokonas PS, Landsberg L, Weiss ST. Uric Acid and coronary heart disease risk: evidence for a role of Uric Acid in the obesity-insulin resistance syndrome: the normative aging study. Am J Epidemiol. 1995;142(3):228-94. [Link] [DOI:10.1093/oxfordjournals.aje.a117634]
14. Lehto S, Niskanen L, Ronnemaa T, Laakso M. Serum Uric Acid is a strong predictor of stroke in patients with non-insulin-dependent Diabetes mellitus. Stroke. 1998;29(3):635-9. [Link] [DOI:10.1161/01.STR.29.3.635]
15. Loh KC, Leow MK. Current therapeutic strategies for type 2 diabetes mellitus. Ann Acad Med Singap. 2002;31(6):722-9. [Link]
16. Low PA, Nickander KK, Tritschler HJ. The roles of oxidative stress and antioxidant treatment in experimental diabetic neuropathy. Diabetes. 1997;46(2):538-42. [Link] [DOI:10.2337/diab.46.2.S38]
17. Macdonald PE, Wheeler MB. Voltage-dependent K(+) channels in pancreatic beta cells: role, regulation and potential as therapeutic targets. Diabetologia. 2005;46(8):1046-62. [Link] [DOI:10.1007/s00125-003-1159-8]